Final Provisional Source Ingest Notes

Scope

This audit records the final cleanup pass that promoted all remaining provisional source notes to focused source-reviewed mode. The pass intentionally distinguishes durable synthesis from orientation/context routing.

Sources Reviewed

• Development, differentiation, and diversity of ILCs: trend digest, source spine, taxonomy background.
• Evolution of ILCs: trend digest and source spine.
• ILCs as major players in inflammatory diseases: trend digest and source spine.
• Regulatory ILCs control innate intestinal inflammation: adaptive/regulatory context with gut label.
• ILC 10 years on: trend digest and source spine.
• ILC diversity, plasticity, and unique functions: trend digest, ILC2/ILC3 taxonomy guardrails.
• Human mucosal ILC3-ILC1-like diversity spectrum: ILC3 taxonomy and boundary-state context.
• Helper-like ILCs in humans and mice: trend digest and taxonomy guardrails.
• Skin-resident ILCs converge on pathogenic effector state: trend/plasticity comparator only.
• ILC3 recruitment and activation promote antitumor immunity: future tumor-immunity context.
• ZBTB46 defines and regulates protective intestinal ILC3s: ILC3 regulation and identity context.
• Context-dependent ILC3 roles in mucosal protection: ILC3 context/guardrail source.
• ILC2s promote inhibitory synapse development and social behavior: extrapulmonary boundary context only.
• Circadian circuits sustain RORgammat-associated ILC3 plasticity control: ILC3 regulation context.
• Enteric GABAergic neuron-derived GABA sustains ILC3 homeostasis via Igfbp7: ILC3 gut neuroimmune regulation context.
• ILC3 RANK signaling promotes tuft-cell hyperplasia and anthelmintic immunity: ILC3 gut epithelial crosstalk context.

Knowledge-Layer Policy

• Broad reviews were integrated mainly into reading routes and source spines.
• Gut, skin, nervous-system, tumor, and other extrapulmonary sources were kept tissue-labeled.
• Primary extrapulmonary sources were used as mechanism or taxonomy context only where they naturally improved ILC2/ILC3 interpretation.
• No source was promoted as direct lung evidence unless its own context supported that claim.

Status

• Date: 2026-05-05
• Sources promoted: 16
• Remaining provisional source pages expected after refresh: 0